Share:
Share this content in WeChat
X
Experience Exchanges
MRI features of tumor like inflammatory demyelinating disease
LI Hong-ying 

DOI:10.3969/j.issn.1674-8034.2015.12.011.


[Abstract] Objective: To investigate the MRI features of the tumor like inflammatory disease in the brain.Materials and Methods: The MR imaging performances of tumor like inflammatory demyelinating disease in eight eases were retrospectively analyzed and their imaging features were summarized.Results: MRI showed localized mass lesions, and showed long T1, T2 signal lesions in eight cases. Enhancement scanning displayed varying degrees of enhancement, 2 cases showed complete ring-like enhancement, 4 cases showed incomplete opening of the ring-like enhancement, and 2 cases showed patchy enhancement; DWI phase was slightly high in early stage. 6 patients were treated with hormone conservative treatment for 4 weeks, the review of the lesions were reduced and did not have any obvious enhancement. 2 cases were surgical treated by preoperative misdiagnosis.Conclusion: MRI examination of the tumor like inflammatory disease of the diagnosis has a certain characteristic, and MRI can monitor outcomes of lesions.
[Keywords] Demyelinating diseases;Magnetic resonance imaging;Diffusion magnetic resonance imaging

LI Hong-ying* Department of MR, Heze Municiple Hospital, Heze 274031, China

*Correspondence to: Li HY, E-mail: lhying666@163.com

Conflicts of interest   None.

Received  2015-10-05
Accepted  2015-11-10
DOI: 10.3969/j.issn.1674-8034.2015.12.011
DOI:10.3969/j.issn.1674-8034.2015.12.011.

[1]
李瑛,李咏梅.瘤样脱髓鞘病变的影像学研究进展.磁共振成像, 2015, 6(3): 225-229.
[2]
刘怀军,李晖,梁莹,等.脑多发性硬化几个MRI征象的再复习及其意义.磁共振成像, 2011, 2(4): 260-263.
[3]
聂婷婷,沈智威,耿宽,等. Cuprizone诱导的C57BL/6小鼠脱髓鞘模型的T2WI及DTI研究.磁共振成像, 2014, 5(6): 467-472.
[4]
Tanaka K, Tanaka M, Brain Nerve. Differential diagnosis of neuromyelitis optica spectrum disorders. Brain and nerve, 2010, 62(9): 953-960.
[5]
Matsuoka T, Matsushita T, Kawano Y, et al. Heterogeneity of aquaporin-4 autoimmunity and spinal cord lesions in multiple sclerosis in Japanese. Brain, 2007, 130(5): 1206-1223.
[6]
Jarius S, Franciotta D, Bergamaschi R. Immunoglobulin M antibodies to aquaporin-4 in neuromyelitis optica and related disorders. Clin Chem Lab Med, 2010, 48(5): 659-663.
[7]
Levin MH, Bennett JL, Verkman AS. Optic neuritis in neuromyelitis optica. Prog Retin Eye Res, 2013, 36(2):159-171.
[8]
Kang ES, Min JH, Lee KH, et al. Clinical usefulness of cell-based indirect immunofluorescence assay for the detection of aquaporin-4 antibodies in neuromyelitis optica spectrum disorder. Ann Lab Med, 2012, 32(5): 331-338.
[9]
Jarius S, Wildemann B. Aquaporin-4 antibodies (NMO-IgG) as a serological marker of neuromyelitis optica: a critical review of the literature. Brain Pathol, 2013, 23(6): 661-683.
[10]
Yoshimura S, Isobe N, Matsushita T. Distinct genetic and infectious profiles in Japanese neuromyelitis optica patients according to anti-aquaporin 4 antibody status. J Neurol Neurosurg Psychiatry, 2013, 84(1): 29-34.
[11]
Yang CS, Zhang DQ, Wang JH. Clinical features and sera anti-aquaporin 4 antibody positivity in patients with demyelinating disorders of the central nervous system from Tianjin. CNS Neurosci Ther, 2014, 20(1): 32-39.
[12]
Misu T, Fujihara K, Itoyama Y. Neuromyelitis optica and anti-aquaporin 4 antibody-an overview. Brain Nerve, 2008, 60(5): 527-537.
[13]
Lana-Peixoto MA, Callegaro D. The expanded spectrum of neuromyelitis optica: evidences for a new definition. Arq Neuropsiquiatr, 2012, 70(10): 807-813.

PREV Magnetic resonance imaging and ultrasound in evaluation of microwave ablation therapy for breast benign nodules
NEXT The feasibility of IDEAL-IQ quantitative evaluation of vertebral fat fraction content in rabbit models of diabetes mellitus
  



Tel & Fax: +8610-67113815    E-mail: editor@cjmri.cn